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Vinpocetine reduces diclofenac-induced acute kidney injury through inhibition of oxidative stress, apoptosis, cytokine production, and NF-κB activation in mice - ScienceDirect
Pharmaceutical Society of Zambia - Diclofenac is not a sweet 🍭. It is a potential poison when abused. Normal kidneys produce a hormone called prostaglandins that are used to protect the kidney
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Diclofenac induced gastrointestinal and renal toxicity is alleviated by thymoquinone treatment - ScienceDirect
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Ameliorative effects of N‐acetyl cysteine on diclofenac‐induced renal injury in male rats based on serum biochemical parameters, oxidative biomarkers, and histopathological study - Nouri - 2019 - Journal of Food Biochemistry -
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A study on the toxic effect of different doses of Diclofenac sodium on the development of the kidney in the postnatal period | PDF
Pharmacist Who Cares - Diclofenac is not a sweet 🍭. It is a potential poison when abused. Normal kidneys produce a hormone called prostaglandins that are used to protect the kidney from
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Histological features of mice kidney after treatment with compounds I,... | Download Scientific Diagram
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Mitigative effect of caffeine against diclofenac-induced hepato-renal damage and chromosomal aberrations in male albino rats | BMC Complementary Medicine and Therapies | Full Text
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Protective effect of curcumin on the kidney of diclofenac sodium-challenged mice: apoptotic, redox potential and histopathological outcomes | The Journal of Basic and Applied Zoology | Full Text
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Thymoquinone alleviates mitochondrial viability and apoptosis in diclofenac-induced acute kidney injury (AKI) via regulating Mfn2 and miR-34a mRNA expressions | SpringerLink
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